mucosa信息详情
n.[解剖]粘膜
antral mucosa───窦黏膜
fibril mucosa───原纤维粘膜
buccal mucosa───颊粘膜；颊黏膜
The methods were applied to study the face of tunica mucosa of the Trionyx sinensis.───该方法用于研究中华鳖消化道粘膜表面。
Objective To study clinical presentations and complications of prolapse of gastric mucosa.───目的探讨胃黏膜脱垂症的临床表现及其并发症。
Enterogastrone is a hormone liberated by the upper intestinal mucosa.───肠泌素是一种由上肠粘膜释放的激素。
A microscopic comparison of normal colonic mucosa on the left and that of an adenomatous polyp (tubular adenoma) on the right is seen here.───左侧为正常结肠黏膜，右侧为腺瘤性息肉（管状腺瘤）。
A microscopic comparison of normal colonic mucosa on the left and that of an adenomatous polyp on the right is seen here.───图示的是显微镜下所见的形态学变化，在左侧为正常的结肠黏膜，右侧为息肉状腺瘤的镜下形态学改变。
However, rectal absorption is often irregular and incomplete, and many drugs cause irritation of the rectal mucosa.───但直肠的吸收常不规则，不完全，而且许多药物对直肠粘膜具有刺激性。
Although adhesion of HP is a crucial progress in pathogenicity, the adhering ability of HP to stomach mucosa is not the only key factor.───在致病性中，粘附固然是首要条件，但并非引起胃粘膜病变的唯一关键因素。
The mucosa of parasitic positions thickened obviously to be gyrus -like and was covered with mucus, and slight hemorrhage was observed.───寄生部位黏膜增厚，呈脑回样增生，颜色灰白，偶见散在点状出血现象，表面附有较多黏液。
In the control group, there was no damage to the gastric mucosa , it integrity, no congestion, edema, ulcers, bleeding and other symptoms.───对照组中的胃粘膜没有发生任何损害，胃粘膜光华、完整，无充血、水肿、溃疡、出血等症状发生。
The inflammatory cells were absent in the colonic mucosa of Crohn's disease patients examined in remission.
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We showed that luminol enhanced chemiluminescence from colonic mucosa biopsy specimens is increased in patients with ulcerative colitis.
Discussion Phagocytic cells are found in the intestinal mucosa; they are heterogeneous immunohistochemically in both normal and inflamed mucosa.
In some experiments the mucosa was completely devoid of mast cells.
The normal gastric mucosa can prevent bypass diffusion of potentially noxious substances from the gastric lumen.
These cells infiltrate into the colonic mucosa and are abundant during the active phase of colitis.
As a result, the inflamed duodenal mucosa may then become susceptible to acid and this may ultimately lead to ulceration.
Occasional islands of regenerative mucosa were present.