paclitaxel信息详情
n.紫杉醇
Objective to evaluate the relation of dose intensity and efficacy, toxicity in advanced breast cancer treated with paclitaxel as a single agent.───目的分析单药紫杉醇治疗晚期乳腺癌剂量强度与疗效和毒性的关系。
Another 54 people did not receive paclitaxel in any form.───另外54人未接受任何形式的紫杉醇。
Revascularization rates were similar with the sirolimus - and paclitaxel-eluting stents in the two trials, at 5.6% versus 5.3%, respectively.───再次血运重建率在西罗莫司和紫杉醇的两个临床试验类似，分别为5.6%和5.3%。
However, sirolimus stents fared better than paclitaxel stents in terms of MI, target vessel revascularization, and late thrombosis.───在心梗、靶血管的血运重建和晚期血栓文面，西罗莫司支架优于紫杉醇支架。
They also found that this synergy occurred with trastuzumab and either paclitaxel or vincristine.───他们发现这种协同作用也发生在脂质体和长春新碱或紫杉醇。
The total effective rate of acupuncture on the treatment of paclitaxel induced sensory neuropathy is higher than that of oxaliplatin.───针刺治疗紫杉醇引起的感觉神经障碍较奥沙利铂的总有效率高。
Stent thrombosis after 1 year was more common with both sirolimus-eluting stents and paclitaxel-eluting stents than with bare-metal stents.───与裸金属支架组相比，1年后雷帕霉素和紫杉醇药物涂层支架组更容易发生血栓。
The overall response rate (ORR) tended to be higher with both weekly nab-paclitaxel regimens than docetaxel by independent radiology review.───通过独立的放射学复查，蛋白结合型紫杉醇单周方案的总有效率（ORR）要比紫杉醇高。
Animal experiment proved that compound paclitaxel phospholipid dispersing substance improves bioavailability of paclitaxel.───动物试验证实，复方紫杉醇磷脂分散物显著提高了紫杉醇的口服生物利用度。
Results The C13 side chain of paclitaxel was obtained with the chemical yield of 41% and ee. of 99% .
The electrochemical behavior of paclitaxel investigated at a hanging mercury drop electrode indicated that the electrode reduction process was quasi-reversible with adsorptive characteristics.
The cytotoxic effect of paclitaxel liposome on human lung adenocarcinoma cell line SPC-A1 in vitro was detected by thiazolyl blue(MTT) assay.
Scientists are now trying to mimic natural paclitaxel, so the trees will not have to die.
The results demonstrate that paclitaxel and daunorubicin can form intermolecular hydrogen bonds.
Paclitaxel can induce apoptosis in C6 cell, apoptosis is correlated nearly to the block in G2-M phase, apoptosis is induced in C6 cell after cells blocked in G2-M phase for some times.
Paclitaxel prevents cancer cells by binding to cell components called microtubules.
Then, paclitaxel is extracted from the bark in a complicated process.
Pneumonitis related to treatment with paclitaxel.