neuronal信息详情
adj.[解剖]神经元的
neural foraminal stenosis───神经孔狭窄
endoneurium quizlet───神经鞘膜
aponeurotic tear───腱膜撕裂
endoneurium def───神经内膜
endoneurium meaning───神经内膜意义
depolarizing neuromuscular blockers───去极化神经肌肉阻滞剂
aortic aneurysm───[内科]主动脉瘤；织脉瘤；主动脉动脉瘤
dor neuropatica───神经病理性疼痛
inserts for neuroma───神经瘤插入物
aponeurotic fiber───腱膜纤维
Like many other agents that affect neuron firing, adenosine must first bind to specific receptors on neuronal membranes.───像许多其他影响神经元放电的物质一样，腺苷必须首先与神经元膜上的特定受体结合。
This physiological rig amplifies neuronal spikes from biological specimens.───这款生理学装置就可放大生物标本的神经元峰电位。
neuronal imbalance in the brain of the infant caused seizures, prompting surgeons to remove the enlarged hemisphere—a treatment that has proven successful in similar cases.───婴儿大脑的神经元失衡导致癫痫发作，促使外科医生切除了扩大的大脑半球——这种治疗在类似病例中被证明是成功的。
At this point, ~85% of these upper motor neuronal axons decussate. These fibers then descend as the lateral corticospinal tract.───至此，有大约85%的上位运动神经元轴突交叉成十字形及如皮质脊髓侧束般下降。
As such, the cellular and molecular mechanisms of opiate-induced neuronal damage have been a focus of extensive research interest.───因此，近些年阿片类药物神经损害的细胞和分子机制已经引起人们的广泛关注。
The animals that had not run before moving in with the mean mice showed much less neuronal activity in these portions of the brain.───那些在放入笼子里前没有跑动的普通老鼠，它们大脑的那个部分显示出非常少的神经活动。
Though the neuronal circuits rarely change, the receptors are constantly being recycled and presented again on the surface.───尽管神经元电路很少改变，但感应器被不断地循环利用，并再次出现在表层。“每个细胞都在不断地自我重建。”
Glial communication is controlled by calcium influxes , just as neuronal communication is.───神经胶细胞之间的沟通是由钙离子的流入来控制，就像神经元之间的沟通一样。
As such, neuronal injury may explain the histopathologic changes that have been observed following chronic rotator cuff tears.───神经损伤可以解释慢性肩袖撕裂病例中观察到的组织病理改变。
Mutations in pros alter neuronal fates and delay or prevent pioneering of motor nerves during embryogenesis.
Intelligence related to neuronal circuits in cortex of brain.
Consider the physiologist's intuition that an increased neuronal firing frequency explains increased intensity of experience.
Our clinical findings suggest that the neuronal disorder may be compensated to some degree because the intestines are richly innervated.
Pain in man requires the presence of distributed neuronal circuits rather than a single locus.
These measurements correlate with neuronal cell count and show a relative reduction of neurons in the epileptogenic hippocampus.
In addition, the neuronal representation and control of complex innate behavioural repertoires of strategic importance is based here.
Brain injury may lead to neuronal cell necrosis.
Many trophic factors can influence axonal growth and neuronal survival during nervous system development and in the regeneration of peripheral nerves.