pepsinogen信息详情
n.胃蛋白酶原
The impressively named chief cells secrete pepsinogen which, when it mixes with stomach acid, becomes an enzyme called pepsin.───而令人印象深刻的主细胞则分泌胃蛋白酶原，当它与胃酸混合时，就形成一种酶，叫做胃蛋白酶。
In this study, the ontogeny of pepsinogen producing cells in rat fundic glands was studied by in situ hybridization using a digoxigenin-labeled RNA probe.───本研究采用地高辛标记的RNA探针，用原位杂交的方法研究了大鼠胃底腺中胃蛋白酶产生细胞的个体发育。
AIM: To explore the matching degree of in situ expression and serum level of pepsinogen C (PGC) in different gastric mucosal biopsies, and to evaluate its value in the diagnosis of gastric cancer.───目的：通过对不同类型胃黏膜活检组织胃蛋白酶原C（PGC）的表达和血清胃蛋白酶原含量的检测，研究二者的匹配程度及其在胃癌筛查与诊断中的价值。
Pepsinogen combine with fuji intelligent color enhancement can improve the rate of early gastric cancer diagnosis.───胃蛋白酶原联合放大染色内镜可提高早期胃癌诊断率。
Serum pepsinogen concentration as an index for Helicobacter pylori eradication───血清胃蛋白酶原含量作为幽门螺杆菌除菌疗效判定指标的研究
Variation in serum pepsinogen concentration before and after Helicobacter pylori eradication───幽门螺杆菌根除前后血清胃蛋白酶原含量变化
Content Detection of Pepsinogen and its Application in the Diagnosis of Disease of Stomach───胃蛋白酶原含量检测及其在胃疾病诊治中的应用
Studies on the cut-off value of serum pepsinogen abnormality for screening chronic atrophic gastritis and gastric carcinoma───胃癌高发区居民血清胃蛋白酶原水平及异常标准的研究
Correlation of serum pepsinogen level and gastric mucosal changes of residents in the high incidence area of gastric cancer───胃癌高发区居民血清胃蛋白酶原水平与胃黏膜病变的关系
In these conditions serum pepsinogen C provides additional diagnostic information, especially when expressed as pepsinogen A:C ratio.
Decreased concentrations of activated pepsinogen were found in H pylori positive patients only. Duodenal biopsies were tested in 76 patients.
High values of serum pepsinogen A have also been found during omeprazole treatment.
From these findings, it can be suggested that pepsinogen genes are involved in the pathogenesis of peptic ulcer.
Decreased concentrations of activated pepsinogen were found in H pylori positive patients only.
The high pepsinogen C concentrations in patients with duodenal ulcer are probably related to H pylori related chronic active gastritis.
Plasma pepsinogen levels are above the normal of 1.0 i.u. tyrosine and usually exceed 2.0 i.u. in sheep with heavy infections.
This results in a failure to activate pepsinogen to pepsi and so denature proteins.
Activated pepsinogen was significantly reduced in the stomach of H pylori positive patients only.